The corona virus was first identified in 1960 and has only recently been linked to flu-like symptoms. This is surprising, because until 2001, the corona virus was considered a simple virus. However, in 2002, an outbreak in Guangdong province, China, prompted severe respiratory syndrome and caused a high mortality rate in more than a thousand patients. This caused scientists to re-examine their understanding of the virus and its transmission mechanisms.
Children’s coronavirus
After nearly two million children got sick in December, the Centers for Disease Control and Prevention (CDC) approved a vaccine for children to prevent the disease. The Pfizer-BioNTech vaccine protects children from the coronavirus, which has been responsible for nearly 2 million childhood cases of illness worldwide. Millions of families have been anxiously awaiting this vaccine, which could mean returning to school or work for kids.
While the disease in children is relatively uncommon compared to adults, the symptoms are similar between the two. However, some children are more likely to develop the disease if they have certain medical conditions or are very young. In addition, children with certain medical conditions, such as asthma, may have a more severe reaction to this illness than healthy children. Infants younger than 12 months may be particularly susceptible, and some children may develop a multisystem inflammatory syndrome requiring medical attention.
Middle East respiratory syndrome coronavirus
The novel betacoronavirus known as MERS is the culprit behind a massive outbreak of severe respiratory disease in Saudi Arabia. It is responsible for the high case fatality rate associated with the condition. Despite the severity of the disease, the virus does not always cause the typical symptoms of pneumonia and shortness of breath. Despite the risk of contracting the disease, the Saudi authorities have already implemented extensive measures to prevent the spread of MERS.
After being isolated from a single human in 2012, the new virus has spread rapidly, with the number of cases in the region now exceeding two thousand. The region remains the most affected, with more than two thousand cases reported in 2018 alone. Despite the high mortality rate, there are no known drugs or vaccines for the disease. However, a large body of research has been conducted on the virus’ structure and pathophysiology.
Novel strain of coronavirus
The novel strain of coronavirus (COVID-19) has been confirmed in the United States, causing respiratory illness in humans and causing more than 693,000 infections worldwide. This strain first emerged in Wuhan, China, and has sickened hundreds and even killed 18 people. Nurses are on the front lines of the effort to contain this outbreak, as COVID-19 is very contagious. It is also contagious before symptoms appear and can easily spread from person to person.
The novel coronavirus was first associated with severe respiratory illness in late 2019 and is now causing deaths and infections around the world. It is a member of the coronavirus family and has been linked to several diseases, including the 2003 SARS outbreak and the Middle East Respiratory Syndrome (MERS), which began in Saudi Arabia in 2012.
Transmission mechanisms
The study of Corona virus transmission has been controversial, but advances in next-generation sequencing technology have made it possible to identify thousands of novel viral sequences from human and animal populations. However, there is limited research on the transmission mechanisms of this virus. Previously, the lack of tools to predict zoonotic virus outbreaks has limited our ability to predict the development and spread of disease. Developing a better understanding of this virus’s transmission mechanisms will help prevent future outbreaks and maintain global health and economic stability.
The first step of Corona virus infection is the attachment of the viral spike protein to a host cell’s receptor. This attachment is necessary for the virus to infect the host cell, but it is also the most difficult aspect of the infection. In addition to triggering the virus’s replication, the spike protein also fuses the host cell’s membrane and releases the virus’s genome. The spike protein consists of a large ectodomain and receptor-binding subunits.
